Scar formation is a major source of morbidity in surgical patients and is a commonly encountered issue in the fields of Plastic and Burn surgery. At present there are limited available treatment strategies to prevent and optimise scar formation. In contrast to adult wounds, foetal wounds sustained in early gestation heal without scar. The potential for scarless healing appears to be intrinsic to the foetal skin itself, as opposed to the foetal environment, as confirmed in multiple experiments. Scarless healing only appears to occur in early gestation as foetal wounds sustained after 24 weeks gestation have been shown heal with scarring. The exact mechanism of scarless foetal wound healing remains largely unknown but there are multiple identified factors that are thought to contribute. Collagen content and type, hyaluronic acid, inflammatory cells, proinflammatory cytokines, fibroblasts and the extracellular matrix have all been implicated in the process of foetal wound healing. This review will focus on and review the differences between foetal and postnatal skin that contribute to scarless healing and provide a summary of the literature to date.